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Tex,
I'm sorry to read about the loss of your uncle. My father's brother is still living at age 90, albeit with dementia. When I see him, I am reminded of my father who died at age 67. Hopefully you will live as long as your uncle, or longer.
I used to look younger than my years also, even after the onset of MC. It wasn't until this summer when I severely restricted my diet and calories, that I began to show my age due to the weight loss. I hope I'm healthier for it.
I've been eating 1900-2000 calories a day in an effort to gain back some of the 32 lbs. I've lost, but so far I've been unsuccessful. I never thought I would be embarrassed about how thin I am.
Rich,
I thought the same as you - that being on this strict of a diet would automatically lower my cholesterol in spite of genetics. I went off my statin and was tested earlier this year. While my count was down - I believe it was 230 - it wasn't low enough to please the doctor and I'm back on the statin. One encouraging note is that a statin supposedly helps with diarrhea.
Gloria
Some Random Thoughts On Longevity And GI System Diseases
Moderators: Rosie, Stanz, Jean, CAMary, moremuscle, JFR, Dee, xet, Peggy, Matthew, Gabes-Apg, grannyh, Gloria, Mars, starfire, Polly, Joefnh
Gloria, this weight business is a mystery to me, and I wish I had some suggestions for you. While I haven't lost as much weight as you, I just can't seem to put it back on, even though I'm a year gluten/dairy/soy/yeast free and feel pretty stable. My pre-MC wight was 130 lbs, I dropped as low as 110 lbs, and now I'm up to 118 lbs but would like another 5 lbs as a "reserve". Even when I deliberately up my calories, it doesn't seem to make much difference. And if I do seem to have put on a couple of pounds, it seems like it only takes a skipped meal, or traveling where I'm not able to eat as much, and the weight that took weeks to put on comes off in a day or two. My guess is that I still have some adsorption issues. Or maybe my metabolism has this new set point.
It's really a struggle to gain weight with our condition, and I know you have been working hard at it. Now that you are getting more stabilized on the Entocort, I hope the weight gain will follow! I'm rooting for you!
Rosie
It's really a struggle to gain weight with our condition, and I know you have been working hard at it. Now that you are getting more stabilized on the Entocort, I hope the weight gain will follow! I'm rooting for you!
Rosie
Our greatest weakness lies in giving up. The most certain way to succeed is always to try just one more time………Thomas Edison
Thanks everyone, for your thoughtful words of sympathy. For us, (his surviving closest relatives), his passing marks the end of an era, because now, we are the "older generation". 
Gloria, like your uncle, for about the last 5 years, my uncle was also plagued with dementia, and he no longer recognized us, but prior to that, he was as sharp as a tack. I suppose dementia is one of the worst risks of living past "normal expectations".
Tex
Gloria, like your uncle, for about the last 5 years, my uncle was also plagued with dementia, and he no longer recognized us, but prior to that, he was as sharp as a tack. I suppose dementia is one of the worst risks of living past "normal expectations".
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
Dear Tex,
I add my condolences and prayers for your Uncle.
And I hope you have his longevity without the dementia. You are already a wise leader, but I see you years from now.... as the wisest of elders - a great and enduring oak tree protecting generations of a growing MC family, guiding the newbies and working with anyone truly willing to find solutions to this humbling condition.
With love, Ant
I add my condolences and prayers for your Uncle.
And I hope you have his longevity without the dementia. You are already a wise leader, but I see you years from now.... as the wisest of elders - a great and enduring oak tree protecting generations of a growing MC family, guiding the newbies and working with anyone truly willing to find solutions to this humbling condition.
With love, Ant
Ant,
I wish I were half as good as the picture you paint. Heck, I'd be happy with a fourth as good. 
Thanks.
Love,
Tex
I wish I were half as good as the picture you paint. Heck, I'd be happy with a fourth as good. Thanks.
Love,
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
Tex,
Your uncle is surely in a better place, happy to be "sharp as a tack" again.
This is an interesting thread on aging, cholesterol, etc. I desperately want to believe that these anti-inflammatory diets we are on will add years of good health to our lifespan. Makes it a nobler cause overall, not just a D preventer.
About cholesterol, both sides of my family have high cholesterol. My dad takes Lipitor and my mom couldn't tolerate it (muscle pain, etc). My dad has a high CRP level and both mom and dad have dangerously low neutropenia. They live on a low-pesticide coffee farm in Guatemala, but I wonder if other pollutants (sugar cane fields and garbage burning nearby, agro-chemicals used in nearby farms, etc) contribute to their condition. Mom recently had 2 operations for subacute subdural hematomas of unknown cause, so there is a bleeding issue there too.
Regarding cholesterol and autoimmune disease, can these diseases use up your cholesterol stores? My level was 187 ten years ago (although I was on birth control pills which could have affected my lipid profile). My level is 150 now, and I understand that's at the too-low cutoff. Can MC and related conditions actually lower your cholesterol? Is cholesterol needed to battle these diseases? I have no shortage of eggs, meat and shellfish in my diet, so with my genes, my level should be much higher.
Your uncle is surely in a better place, happy to be "sharp as a tack" again.
This is an interesting thread on aging, cholesterol, etc. I desperately want to believe that these anti-inflammatory diets we are on will add years of good health to our lifespan. Makes it a nobler cause overall, not just a D preventer.
About cholesterol, both sides of my family have high cholesterol. My dad takes Lipitor and my mom couldn't tolerate it (muscle pain, etc). My dad has a high CRP level and both mom and dad have dangerously low neutropenia. They live on a low-pesticide coffee farm in Guatemala, but I wonder if other pollutants (sugar cane fields and garbage burning nearby, agro-chemicals used in nearby farms, etc) contribute to their condition. Mom recently had 2 operations for subacute subdural hematomas of unknown cause, so there is a bleeding issue there too.
Regarding cholesterol and autoimmune disease, can these diseases use up your cholesterol stores? My level was 187 ten years ago (although I was on birth control pills which could have affected my lipid profile). My level is 150 now, and I understand that's at the too-low cutoff. Can MC and related conditions actually lower your cholesterol? Is cholesterol needed to battle these diseases? I have no shortage of eggs, meat and shellfish in my diet, so with my genes, my level should be much higher.
1987 Mononucleosis (EBV)
2004 Hypomyopathic Dermatomyositis
2009 Lymphocytic Colitis
2010 GF/DF/SF Diet
2014 Low Dose Naltrexone
2004 Hypomyopathic Dermatomyositis
2009 Lymphocytic Colitis
2010 GF/DF/SF Diet
2014 Low Dose Naltrexone
Zizzle,
IMO, High-Sensitivity C-Reactive Protein is the key to monitoring the risk of diseases affected by long-term inflammatory issues. Of course, H-S CRP can only be accurately assessed when no acute inflammatory processes are present.
I'm sure you're familiar with the possible causes of neutropenia and/or aplastic anemia, but have their B-12 and folate levels been checked? Some individuals require not just "normal" levels of these vital vitamins, but relatively high levels, and this insufficiency can certainly lead to these conditions.
Hmmmmmmmmm. You have posed a very interesting question. I'm not aware of any research that has addressed the relationship between total cholesterol level and MC, specifically, but there appears to be a correlation with IBDs in general. Consider this:
Note that the discussion in the last paragraph of that quote, suggests exactly the opposite effect of what would be intuitive for the situation that we're discussing, (IOW, it implies that lowering the cellular cholesterol level would suppress inflammation, not enhance it), but I'm not sure that this applies directly to serum cholesterol levels. Just because serum levels of any given substance are high, (or normal, or low), does not justify the assumption that cellular levels will be high, (or normal, or low), or vice versa. Magnesium is a good example of that, for example. Conventional blood tests for magnesium are pretty much worthless, because serum magnesium levels will remain normal, even though cells may be starving for magnesium. By the time the serum magnesium level shows a significant decline, the cellular level will be extremely low.
Okay, here's how cholesterol is recovered in the intestines, (I'll use some applicable quotes, to save time typing):
http://www.vivo.colostate.edu/hbooks/pa ... /bile.html
Most of the cholesterol recovery, (bile fatty acids), occurs in the terminal ileum. When I had my surgery in February, they removed not only my colon, but also about three and a half inches of my terminal ileum. By the middle of April, (70 days after my surgery), my total cholesterol was down by only about 7 or 8 points from where it normally ranges for me. But from there until the 5th of May, (after only 22 additional days), it had dropped another 20 points, so it was obviously beginning to accelerate the rate of fall. During the next 27 days, it dropped an additional 46 points, (to 145). A month later, it had recovered 21 points, and it seems to have stabilized in that area, (on Oct. 8th, it was 161). Now, I started taking a statin in May, (due to a second TIA in less than a year), but there is no way that a statin can reduce cholesterol levels so drastically, that fast, so obviously my missing terminal ileum was a big factor in this drastic decline. My total serum cholesterol had dropped at least 27 points, before I started taking the statin.
MC primarily affects the ascending colon and the terminal ileum, (in most cases - and this is why biopsies taken by means of a sigmoidscopy can sometimes cause a missed diagnosis). At any rate, inflammation is typically most concentrated in the ascending colon and the terminal ileum, (even though technically, the ileum is not "allowed" to be involved, because the name "colitis" refers to the colon, only). Because of the presence of inflammation in the terminal ileum, it is certainly conceivable that bile fatty acids are very likely malabsorbed, leading to a reduction of total serum cholesterol, whenever MC is active for an extended period of time. To my knowledge, this has never been researched, so this is primarily just a WAEG on my part, but I'll bet a GF cookie that it's basically sound.
Tex
IMO, High-Sensitivity C-Reactive Protein is the key to monitoring the risk of diseases affected by long-term inflammatory issues. Of course, H-S CRP can only be accurately assessed when no acute inflammatory processes are present.
I'm sure you're familiar with the possible causes of neutropenia and/or aplastic anemia, but have their B-12 and folate levels been checked? Some individuals require not just "normal" levels of these vital vitamins, but relatively high levels, and this insufficiency can certainly lead to these conditions.
Hmmmmmmmmm. You have posed a very interesting question. I'm not aware of any research that has addressed the relationship between total cholesterol level and MC, specifically, but there appears to be a correlation with IBDs in general. Consider this:
http://esciencenews.com/articles/2008/0 ... el.diseaseCholesterol modulates sorting of CEA -- implications for inflammatory bowel disease
Published: Thursday, May 1, 2008 - 21:42 in Biology & Nature
2 cells, cholesterol was depleted by a combination of synthesis inhibition and plasma membrane extraction with complexing agents. This led to an increased sorting of CEA to the basolateral surface. Interestingly, polarity was not significantly affected by this approach. The association of CEA to lipid rafts, cholesterol, and sphingolipid-enriched microdomains was inhibited in parallel. This study, performed by a team led by Dr. Robert Ehehalt, is described in a research article to be published on March 14, 2008, in the World Journal of Gastroenterology.
Carcinoembryonic antigen (CEA) is a GPI-anchored glycoprotein that has been suggested to have different functions depending on its expression at the apical or basolateral plasma membrane. Whereas at the apical membrane, it is possible for it to interact with bacteria, basolateral expression might be involved in the activation of suppressor T-cells and thus have anti-inflammatory properties. A decreased expression in the basolateral compartment has been attributed to the pathogenesis of inflammatory bowel disease (IBD).
Note that the discussion in the last paragraph of that quote, suggests exactly the opposite effect of what would be intuitive for the situation that we're discussing, (IOW, it implies that lowering the cellular cholesterol level would suppress inflammation, not enhance it), but I'm not sure that this applies directly to serum cholesterol levels. Just because serum levels of any given substance are high, (or normal, or low), does not justify the assumption that cellular levels will be high, (or normal, or low), or vice versa. Magnesium is a good example of that, for example. Conventional blood tests for magnesium are pretty much worthless, because serum magnesium levels will remain normal, even though cells may be starving for magnesium. By the time the serum magnesium level shows a significant decline, the cellular level will be extremely low.
Okay, here's how cholesterol is recovered in the intestines, (I'll use some applicable quotes, to save time typing):
Bile acids are derivatives of cholesterol synthesized in the hepatocyte. Cholesterol, ingested as part of the diet or derived from hepatic synthesis is converted into the bile acids cholic and chenodeoxycholic acids, which are then conjugated to an amino acid (glycine or taurine) to yield the conjugated form that is actively secreted into cannaliculi.
Hepatic synthesis of bile acids accounts for the majority of cholesterol breakdown in the body. In humans, roughly 500 mg of cholesterol are converted to bile acids and eliminated in bile every day. This route for elimination of excess cholesterol is probably important in all animals, but particularly in situations of massive cholesterol ingestion.
Interestingly, it has recently been demonstrated that bile acids participate in cholesterol metabolism by functioning as hormones that alter the transcription of the rate-limiting enzyme in cholesterol biosynthesis.
Large amounts of bile acids are secreted into the intestine every day, but only relatively small quantities are lost from the body. This is because approximately 95% of the bile acids delivered to the duodenum are absorbed back into blood within the ileum.
http://www.vivo.colostate.edu/hbooks/pa ... /bile.html
Most of the cholesterol recovery, (bile fatty acids), occurs in the terminal ileum. When I had my surgery in February, they removed not only my colon, but also about three and a half inches of my terminal ileum. By the middle of April, (70 days after my surgery), my total cholesterol was down by only about 7 or 8 points from where it normally ranges for me. But from there until the 5th of May, (after only 22 additional days), it had dropped another 20 points, so it was obviously beginning to accelerate the rate of fall. During the next 27 days, it dropped an additional 46 points, (to 145). A month later, it had recovered 21 points, and it seems to have stabilized in that area, (on Oct. 8th, it was 161). Now, I started taking a statin in May, (due to a second TIA in less than a year), but there is no way that a statin can reduce cholesterol levels so drastically, that fast, so obviously my missing terminal ileum was a big factor in this drastic decline. My total serum cholesterol had dropped at least 27 points, before I started taking the statin.
MC primarily affects the ascending colon and the terminal ileum, (in most cases - and this is why biopsies taken by means of a sigmoidscopy can sometimes cause a missed diagnosis). At any rate, inflammation is typically most concentrated in the ascending colon and the terminal ileum, (even though technically, the ileum is not "allowed" to be involved, because the name "colitis" refers to the colon, only). Because of the presence of inflammation in the terminal ileum, it is certainly conceivable that bile fatty acids are very likely malabsorbed, leading to a reduction of total serum cholesterol, whenever MC is active for an extended period of time. To my knowledge, this has never been researched, so this is primarily just a WAEG on my part, but I'll bet a GF cookie that it's basically sound.
Good question. It is known that cholesterol is protective of infection and cancer, in older people, (IOW, research shows that they live longer with serum cholesterol levels classified as high to very high, and the primary difference in markers is lowered infection and cancer rates), but these were longevity studies, and didn't specifically address the mechanisms involved. Probably, though, it does play a role in the control of inflammation, IMO. Doctors are so biased against cholesterol, that they don't want to admit that it's actually good for anything. It's essential, of course, for the production of insulin, and all sorts of other vital body chemicals.Zizzle wrote:Is cholesterol needed to battle these diseases?
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
I googled low cholesterol and IBDs and came across the strangest website:
http://www.biblelife.org/bowel.htm
This guy wrote a book, "Absolute Truth Exposed Volume 1" by Kent R. Rieske
It seems like rantings by a conspiracy theorist, but ironically, it espouses most of the diet recommendations we talk about here, but with a greater emphasis on more meat, no sugar or carbs, and some cheese. This guy seems to know and research alot, but his website won't win over many evidence-minded folk. I'm noticing a trend in all these "expert" websites all recommending variations of the same diet to treat IBD. It's those nuances and variations that we seem to be working out here.
Anyway, a number of sources cite low total cholesterol (especially insufficient HDL) as a common occurence in IBD, so I'll raise you a GF cookie, and agree that it must be due to malabsorption.
http://www.biblelife.org/bowel.htm
This guy wrote a book, "Absolute Truth Exposed Volume 1" by Kent R. Rieske
It seems like rantings by a conspiracy theorist, but ironically, it espouses most of the diet recommendations we talk about here, but with a greater emphasis on more meat, no sugar or carbs, and some cheese. This guy seems to know and research alot, but his website won't win over many evidence-minded folk. I'm noticing a trend in all these "expert" websites all recommending variations of the same diet to treat IBD. It's those nuances and variations that we seem to be working out here.
Anyway, a number of sources cite low total cholesterol (especially insufficient HDL) as a common occurence in IBD, so I'll raise you a GF cookie, and agree that it must be due to malabsorption.
1987 Mononucleosis (EBV)
2004 Hypomyopathic Dermatomyositis
2009 Lymphocytic Colitis
2010 GF/DF/SF Diet
2014 Low Dose Naltrexone
2004 Hypomyopathic Dermatomyositis
2009 Lymphocytic Colitis
2010 GF/DF/SF Diet
2014 Low Dose Naltrexone
-
Gabes-Apg
- Emperor Penguin
- Posts: 8367
- Joined: Mon Dec 21, 2009 3:12 pm
- Location: Hunter Valley NSW Australia
Tex
my father and his brother both died of brain tumours (my dad aged 51 his brother aged 54)
During their childhood and teenage years both of them handled large quantaties of DDT (killing blackberries on the farm)
During her childhood my mother used to play with the pretty rainbows in the puddles around the apple trees (the Aust govt did compulsary spraying of the crops with chemicals in the 1930's/1940s)
given my birth defects (no ligaments holding my bowels in place) and subsequent allergies and health issues, i am pretty sure that a main contributor was the impact of the DDT on their cells, that cause my cells to be a bit less than perfect.
my ponderings are even if i lived a wholesome life (good organic food, no stress) could i have avoided MC? or do my genes and my cells dictate the outcome no matter what.
how many health issues are purely gene related? and how many are the outside influences?
my father and his brother both died of brain tumours (my dad aged 51 his brother aged 54)
During their childhood and teenage years both of them handled large quantaties of DDT (killing blackberries on the farm)
During her childhood my mother used to play with the pretty rainbows in the puddles around the apple trees (the Aust govt did compulsary spraying of the crops with chemicals in the 1930's/1940s)
given my birth defects (no ligaments holding my bowels in place) and subsequent allergies and health issues, i am pretty sure that a main contributor was the impact of the DDT on their cells, that cause my cells to be a bit less than perfect.
my ponderings are even if i lived a wholesome life (good organic food, no stress) could i have avoided MC? or do my genes and my cells dictate the outcome no matter what.
how many health issues are purely gene related? and how many are the outside influences?
Gabes Ryan
"Anything that contradicts experience and logic should be abandoned"
Dalai Lama
"Anything that contradicts experience and logic should be abandoned"
Dalai Lama
Gabes,
Did you have malrotation of the bowel as an infant? How did you find out? My friend's adopted son from Russia had this condition, first evident at 7 yrs old! His bowel was on the opposite side of the body and had to be "attached" to prevent it from migrating all over the place. He is otherwise an extremely healthy boy.
Did you have malrotation of the bowel as an infant? How did you find out? My friend's adopted son from Russia had this condition, first evident at 7 yrs old! His bowel was on the opposite side of the body and had to be "attached" to prevent it from migrating all over the place. He is otherwise an extremely healthy boy.
1987 Mononucleosis (EBV)
2004 Hypomyopathic Dermatomyositis
2009 Lymphocytic Colitis
2010 GF/DF/SF Diet
2014 Low Dose Naltrexone
2004 Hypomyopathic Dermatomyositis
2009 Lymphocytic Colitis
2010 GF/DF/SF Diet
2014 Low Dose Naltrexone
Zizzle,
I agree that there are a lot of similar web sites that contain a fair amount of truth, but they leave a "used car salesman" impression.
FWIW, my HDL has always been low, (near the bottom of the "legal" range), probably due to genetics, and, according to an Enterolab test, I still had a malabsorption problem, 3 and a half years after starting the GF diet, and 2 years after achieving remission.
Tex
I agree that there are a lot of similar web sites that contain a fair amount of truth, but they leave a "used car salesman" impression.
FWIW, my HDL has always been low, (near the bottom of the "legal" range), probably due to genetics, and, according to an Enterolab test, I still had a malabsorption problem, 3 and a half years after starting the GF diet, and 2 years after achieving remission.
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
Gabes,
I have to agree that most of your health issues could probably be traced to hereditary influences.
In general, genes predispose to certain conditions, (that is, they are necessary conditions, but they are not sufficient, by themselves, to actually cause the disease to develop). Most genes are triggered by environmental conditions, during our lifetimes, (IOW, they provide the "sufficiency" requirement that is needed to actually trigger the disease). It's impossible to predict the outcome, because so many "events" change our genetics, which then changes the potential for other possible subsequent events. Viruses, for example, can change our genes.
Tex
I have to agree that most of your health issues could probably be traced to hereditary influences.
In general, genes predispose to certain conditions, (that is, they are necessary conditions, but they are not sufficient, by themselves, to actually cause the disease to develop). Most genes are triggered by environmental conditions, during our lifetimes, (IOW, they provide the "sufficiency" requirement that is needed to actually trigger the disease). It's impossible to predict the outcome, because so many "events" change our genetics, which then changes the potential for other possible subsequent events. Viruses, for example, can change our genes.
Tex
It is suspected that some of the hardest material known to science can be found in the skulls of GI specialists who insist that diet has nothing to do with the treatment of microscopic colitis.
-
Gabes-Apg
- Emperor Penguin
- Posts: 8367
- Joined: Mon Dec 21, 2009 3:12 pm
- Location: Hunter Valley NSW Australia
zizzle
from what my mother told me i had digestion issues from birth (colic fluctuation of D and C, waking alot during the night crying)
during my teenage years i had many episodes of being in extreme cramping pain and fatigues issues, on two instances where it was quite bad i went to hospital with suspect apendicitis, they would put me on a drip and bed rest and 2 days later the pain would go.
they did many scans of my ovaries etc and doctors labelled it 'growing pains'
(noting at this stage i had not menstrated due to PCOS i did not start menstration until age 17)
into my early twenties, again i had further episodes (nausea, vomitting, extreme pain) some visits to emergency dept and no clear reason why, with rest and minimal eating would abate
i did not get the exact diagnosis until the 2nd surgery when i was 25.
during this surgery (bowel was 90% twisted) they said the bowel contision was because i had no ligaments holding the large bowel in place the small intestine was quite damaged with adhesions due to the migration everywhere around my body
since childhood and even now, if the bowel is a bit inflammed and i turn quickly in bed it 'catches' (ie i get a sharp jabbing pain) obviously waking me up, i have to turn back slowly and it will be a bit sore for a day or two. the bowel is also adhered to one of my ovaries has been there since 1997 and subsequent ultrasounds to check the PCOS situation have confirmed it is still adhered there.
that is fantastic for your friends that they did get it 'attached' and he is very healthy. (i asked the surgeon if they could cable tie it to my rib cage he said no...)
your friends son is the first time i have heard of this occuring in another person!
in the hospital i had lots of doctors and students quizzing me about my medical history as it was a rare situation
from what my mother told me i had digestion issues from birth (colic fluctuation of D and C, waking alot during the night crying)
during my teenage years i had many episodes of being in extreme cramping pain and fatigues issues, on two instances where it was quite bad i went to hospital with suspect apendicitis, they would put me on a drip and bed rest and 2 days later the pain would go.
they did many scans of my ovaries etc and doctors labelled it 'growing pains'
(noting at this stage i had not menstrated due to PCOS i did not start menstration until age 17)
into my early twenties, again i had further episodes (nausea, vomitting, extreme pain) some visits to emergency dept and no clear reason why, with rest and minimal eating would abate
i did not get the exact diagnosis until the 2nd surgery when i was 25.
during this surgery (bowel was 90% twisted) they said the bowel contision was because i had no ligaments holding the large bowel in place the small intestine was quite damaged with adhesions due to the migration everywhere around my body
since childhood and even now, if the bowel is a bit inflammed and i turn quickly in bed it 'catches' (ie i get a sharp jabbing pain) obviously waking me up, i have to turn back slowly and it will be a bit sore for a day or two. the bowel is also adhered to one of my ovaries has been there since 1997 and subsequent ultrasounds to check the PCOS situation have confirmed it is still adhered there.
that is fantastic for your friends that they did get it 'attached' and he is very healthy. (i asked the surgeon if they could cable tie it to my rib cage he said no...)
your friends son is the first time i have heard of this occuring in another person!
in the hospital i had lots of doctors and students quizzing me about my medical history as it was a rare situation
Gabes Ryan
"Anything that contradicts experience and logic should be abandoned"
Dalai Lama
"Anything that contradicts experience and logic should be abandoned"
Dalai Lama
Gabes,
Interestingly, my friend is from Australia, but she lives here. From what I recall, his doctor said his bowels were happily lodged in an alternate location until physical activity, probably swimming, dislodged them. He had several days, maybe weeks, of belly pain before they discovered it. I think they also said it is a birth defect that happens at a specific time during early pregnancy. His only other issue (so far) is nighttime bedwetting. I
I suppose it could be related, but they haven't made that connection. Let me know if you'd like to get connected to my friend for more info.
Interestingly, my friend is from Australia, but she lives here. From what I recall, his doctor said his bowels were happily lodged in an alternate location until physical activity, probably swimming, dislodged them. He had several days, maybe weeks, of belly pain before they discovered it. I think they also said it is a birth defect that happens at a specific time during early pregnancy. His only other issue (so far) is nighttime bedwetting. I
I suppose it could be related, but they haven't made that connection. Let me know if you'd like to get connected to my friend for more info.
1987 Mononucleosis (EBV)
2004 Hypomyopathic Dermatomyositis
2009 Lymphocytic Colitis
2010 GF/DF/SF Diet
2014 Low Dose Naltrexone
2004 Hypomyopathic Dermatomyositis
2009 Lymphocytic Colitis
2010 GF/DF/SF Diet
2014 Low Dose Naltrexone